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Murray Walker: Incredible!: A Tribute to a Formula 1 Legend

為了解決Euphoria的問題，作者Hamilton, Maurice 這樣論述：

A celebration of the extraordinary life of legendary commentator Murray Walker, with tributes from key figures in Formula 1 and motorsport. Murray Walker was the voice of Formula One, matching the thrill of the track with his equally fast-paced and exhilarating commentary, delivering the euphoria of

motor racing to millions. Commentating on his first grand prix for the BBC at Silverstone in 1949, Murray’s broadcasting career spanned over fifty years. His natural warmth and infectious enthusiasm won great affection with audiences, whilst his passion and knowledge of motorsport allowed him to h

one his instinctive presenting style into a craft. When Murray passed away in March 2021, tributes came flooding in from every corner of the sporting world. This book, compiled by Murray’s great friend and colleague Maurice Hamilton, celebrates the extraordinary life of this truly legendary man. Wi

th contributions from drivers and industry figures, and many friends from the world of motorsport and beyond, Incredible! combines fond memories, never-before-told stories and famous Murrayisms with reflections on the highlights of a life lived at full throttle. Maurice Hamilton has been covering

Formula 1 as a freelance journalist since 1977. He has attended more than 450 Grands Prix, including every race since 1984. The author of 19 books, Hamilton also commentates on the Grands Prix for BBC Radio 5 Live.

Euphoria進入發燒排行的影片

運用高通量技術偵測基因變異性建立注意力不足過動症之輔助檢測平台

為了解決Euphoria的問題，作者孫葶軒 這樣論述：

中文摘要 iAbstract iiList of Figures viList of Tables viiiChapter 1 Introduction 11.1. Neurodevelopmental disorders 11.2. Attention-deficit/hyperactivity disorder (ADHD) 51.2.1. Symptoms and diagnosis 51.2.2. Comorbidities and ADHD 61.2.3. ADHD etiology and

risk factors 91.2.4. ADHD treatments 111.2.5. Adult ADHD (aADHD) 151.3. Biological background 151.3.1. Variant number of tandem repeat polymorphisms (VNTRs) 161.3.2. Single nucleotide polymorphisms (SNPs) 231.4. Related worked 241.4.1. The ADHDgene database

241.4.2. The PharmGKB database 241.4.3. Existing genetic tests for ADHD 261.4.4. Machine learning based prediction for diagnosis 281.5. Motivation and research goals 301.5.1. Improve the diagnostic process in ADHD 301.5.2. Provide the evidence of racial/ethnic dif

ferences may alter disease-related genetic patterns 331.5.3. Build a predictor to assess the risk of developing ADHD 34Chapter 2 Material and Methods 352.1. Clinical subject recruitment 352.2. Variant Candidate Selection 352.3. DNA extraction method 422.4. Genotyp

e analysis of VNTRs 422.5. Genotype analysis of SNPs 452.6. Bioinformatics 512.6.1. Target SNPs genotype calling 512.6.2. Calculate risk score for each ADHD-associated loci in our cohort 512.6.3. Data set and quality control for the machine learning process 522.6.4.

Feature relevance learning with random forest 52Chapter 3 Results 543.1. Demographic Characteristics 543.2. Survey report of the 200 ADHD patients 543.3. Successfully identify the allele/genotype of selected markers in cases and controls 583.4. Gender may also play a

role in associations between VNTRs and ADHD 683.5. Association of VNTRs in the noncoding region of DAT, 5-HTT, and IL-1Ra significantly correlated to ADHD 693.6. Three susceptibility SNPs, namely, rs75732170, rs4680, and rs2717162, are risk factors for ADHD 713.7. Apply machine le

arning technique to build a decision-support tool for ADHD risk prediction 71Chapter 4 Discussion 77Chapter 5 Summary and future works 85References 86

The Liar: How a CIA Double Agent Became the Last Honest Man of the Cold War

為了解決Euphoria的問題，作者Cunningham, Benjamin 這樣論述：

The Cold War meets Mad Men in the form of Karel Koecher, a double agent whose shifting loyalties and over-the-top hedonism reverberated from New York to Moscow In the mid-1970s, the CIA and KGB watched Karel Koecher closely--they were both convinced he was working for the enemy. And they were bot

h right. Traveling with his wife, Hana, Koecher posed as a Czechoslovak asylum seeker and arrived in the US as a Communist sleeper agent. After parlaying a doctorate from Columbia into a job at the CIA, Koecher proceeded to operate as a double agent at the height of the Cold War. Shunning a low pro

file, the Koechers embraced Manhattan’s high life--with cocaine, swinging, and parties emblematic of the times and their penchant for risk. Hana, who was no more than a shy teenager when she arrived, grew into a sophisticated international diamond dealer who relayed messages to Karel’s handlers. Rid

ing a wave of euphoria, the Koechers felt unstoppable. But it was too good to last. Using newly declassified documents, interrogation tapes, and extraordinary firsthand accounts from the Koechers themselves, Cunningham reconstructs their double lives and the fading Cold War, where a strange moral f

og made it hard to know what truth was being fought for, and to what end.

心律不整與失智症風險關聯研究：回溯性研究

為了解決Euphoria的問題，作者黃衍彰 這樣論述：

文獻探討：隨人口老化趨勢，失智症的議題受到關注，早期偵測的意識也逐漸抬頭。大多探討心律不整與失智症的研究將關聯性歸因於心房纖維震顫(atrial fibrillation, AF)－心律不整中最常見的類型。心律不整還包含了其他的類型，且研究證明其他類型的心律不整和失智症之間也存在關聯。目前以亞洲樣本探討心律不整與失智症關聯性的研究較少，且過去台灣研究未將心律不整對於輕度認知缺損(mild cognitive impairment, MCI)患者納入研究當中進行討論。本研究探討台灣中南部地區，心律不整對MCI和失智症發生風險影響的預測。研究方法：本研究使用「秀傳醫療體系－失智症登錄資料庫」進行

分析，以基線為認知正常與MCI參與者，且追蹤後認知狀態維持或惡化者進行分析，研究參與者共分為基線認知正常轉變為MCI或轉變為失智症，並考慮年齡效果，將MCI患者分為74歲以上與75歲以下患者共計四組。本研究透過羅吉斯迴歸，探討心律不整與其他變項對預測轉變發生的影響。研究結果：本研究中，基線認知正常組平均追蹤496.24 ± 274.93天，基線MCI組平均追蹤416.37 ± 218.84天。本研究中在75歲以上且基線MCI的參與者中，心律不整參與者失智症的風險較高。另外，高血壓和妄想症狀預測75歲以上MCI轉變為失智症，使用抗血脂藥物預測74歲以下MCI轉變為失智症，具有高膽固醇血症的75歲

以上MCI以及服用抗高血壓藥物或具有易怒症狀的74歲以下MCI轉變為失智症的風險較低；焦慮症狀預測基線認知正常轉變MCI。討論與建議：本研究發現患有MCI，且有心律不整的75歲以上長者，轉變為失智症的風險較高，且另外發現血壓控制與總膽固醇含量對失智症的影響。可供臨床上健康照護參考，透過適當心律不整的控制與治療，以及心血管風險因子的管理，減少高齡長者轉變為失智症之風險程度。